Mechanisms of increased acid secretion after eradication of Helicobacter pylori infection

E M El-Omar

Research output: Contribution to journalEditorial

21 Citations (Scopus)

Abstract

Eradication of Helicobacter pylori infection leads to recovery of gastric acid secretion in some individuals but the mechanism is not fully understood. In the short term, there is an impressive increase in expression of H+/K+-ATPase pumps without an increase in the number of parietal cells. Longer follow up studies are needed to see if the parietal cell mass eventually recovers.

Original languageEnglish
Pages (from-to)144-146
Number of pages3
JournalGut
Volume55
DOIs
Publication statusPublished - 2006

Keywords

  • PARIETAL-CELLS
  • GASTRIC-ACID
  • INTERLEUKIN-1-BETA
  • CARCINOGENESIS
  • APOPTOSIS
  • CORPUS

Cite this

Mechanisms of increased acid secretion after eradication of Helicobacter pylori infection. / El-Omar, E M .

In: Gut, Vol. 55, 2006, p. 144-146.

Research output: Contribution to journalEditorial

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abstract = "Eradication of Helicobacter pylori infection leads to recovery of gastric acid secretion in some individuals but the mechanism is not fully understood. In the short term, there is an impressive increase in expression of H+/K+-ATPase pumps without an increase in the number of parietal cells. Longer follow up studies are needed to see if the parietal cell mass eventually recovers.",
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AB - Eradication of Helicobacter pylori infection leads to recovery of gastric acid secretion in some individuals but the mechanism is not fully understood. In the short term, there is an impressive increase in expression of H+/K+-ATPase pumps without an increase in the number of parietal cells. Longer follow up studies are needed to see if the parietal cell mass eventually recovers.

KW - PARIETAL-CELLS

KW - GASTRIC-ACID

KW - INTERLEUKIN-1-BETA

KW - CARCINOGENESIS

KW - APOPTOSIS

KW - CORPUS

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